Question that’s half real and half facetious:
if it doesn’t alter your mitochondrial density and cardiovascular function then it’s just a pain reliever right?
When I do my work, still only in 2 1/2 years working out, the limiter always seems to be the actual physiology of energy delivery or cardiac function.
Then again there’s ideas about athletes who can tolerate versus people who convert lactate different ways (Phelps vs Hamilton). Thought I read that Phelps perception of it is very low in terms of RPE wheras his body is full of it. Then others bodies have naturally lower levels.
Just postulating.